Authors: W John Martin
This article extends the concept of stealth adaptation of viruses beyond the avoidance of cell mediated immune recognition due to deletion or mutation of the genes coding for the relatively few virus components normally targeted by the cellular immune system. A fragmented virus genome can incorporate additional cellular and bacterial genetic sequences, while continuing to delete the originating virus sequences. Virus replication and transmission extends to the incorporated and substituted sequences, which have seemingly abandoned their normal locations to essentially become viral. By analogy with deserting or with the switching of allegiance, the term renegade sequences is suggested to describe the incorporated sequences beyond those remaining from the initiating virus. The over expression of certain genetic sequences has been linked to the occurrence of particular types of illnesses, including the development of various cancers. Although yet to be clinically acknowledged, this is an inherent risk of transmissible, genetically altered cellular sequences. Humans have become infected with monkey-derived stealth adapted viruses. The monkey-derived cellular sequences in these viruses can potentially undergo homologous recombination with human germline DNA leading to multigenerational illnesses. These and other major implications of the existence in humans of reformed viruses being disguised as self, and/or as bacteria are discussed in this article.
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