Authors: Graham Wilfred Ewing
In 2020 the author was approached by the editorial team of a medical journal, which specialises in Diabetes and Obesity, to compile and submit for publication a summary of his published work, in particular his/the conclusions and papers re diabetes and obesity.
At the outset of this work/research in the period 2003-2007, and the subsequent compilation of >85 peer-reviewed medical papers, the author sought to demonstrate the relationship between sense perception and pathological onset which was considered to be fundamentally important and significant regarding an understanding of how the brain functions and, in particular, how it regulates the autonomic nervous system and/or physiological systems. Diabetes and Developmental Dyslexia provided the two most significant obvious examples because changes of colour perception are recognised to be associated with the onset and progression of these conditions. Moreover, many researchers have sought, unsuccessfully, to develop a precise understanding of this relationship.
The author based this programme of research and publications on ‘the mathematical model of the relationship between sense perception, brain function, the function of the autonomic nervous system and physiological systems, and changes to cellular and molecular biology’ which had been developed by Grakov IG in the period commencing 1981/2 until the prototype system entered the market in 1997, and the end of his research in 2006.
This particular paper focusses upon the fundamental nature of diabetes and obesity. It incorporates and explains the many different aspects of diabetes which often cannot be satisfactorily explained by contemporary biomedicine e.g. that diabetes is a (multi-) systemic disorder which comprises pathological conditions/disorders which occur in the organs in the physiological system which regulates blood glucose; that problems of blood glucose regulation may occur as a result of pathological onset in other adjacent physiological systems e.g. following a hysterectomy; that each pathology may have genetic origins e.g. T1DM, and non-genetic/phenotypic origins e.g. T2DM; that the brain regulates the stable and coherent function of the physiological systems; and that it does so by a biophysical mechanism.
The author singles out suitable references from the peer-reviewed press to support the conclusions made by the author in this programme of published papers, in particular articles re diabetes and obesity, which have been published since 2007.
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